Molecular mechanisms of hyperthermia-induced apoptosis enhanced by docosahexaenoic acid: Implication for cancer therapy

Keyphrases

• Cancer Therapy 100%
• Induced Apoptosis 100%
• Molecular Mechanism 100%
• Hyperthermia Induced 100%
• Docosahexaenoic Acid 100%
• Apoptosis 37%
• Protein Kinase 25%
• Mitochondria 25%
• Intracellular Reactive Oxygen Species 25%
• U937 Cells 12%
• Mitochondrial Proteins 12%
• Phosphorylation 12%
• Dose Effect 12%
• Combined Treatment 12%
• Induced Cell Death 12%
• HeLa Cells 12%
• Hyperthermia 12%
• Acid Treatment 12%
• Pro-apoptotic 12%
• Caspase-3 Activation 12%
• Cancer Treatment 12%
• Bcl-xL 12%
• Mitochondrial Damage 12%
• Bcl-2 Family Proteins 12%
• Mitochondrial Transmembrane Potential 12%
• Anti-apoptotic Bcl-2 12%
• Caspase Pathway 12%
• Protein Kinase C Activation 12%
• TBid 12%
• Significant Protein 12%

Pharmacology, Toxicology and Pharmaceutical Science

• Malignant Neoplasm 100%
• Hyperthermia 100%
• Docosahexaenoic Acid 100%
• Protein Kinase C 37%
• Reactive Oxygen Metabolite 25%
• Antiapoptotic 12%
• Caspase 12%
• Caspase 3 12%

Neuroscience

• Docosahexaenoic Acid 100%
• Protein Kinase C 37%
• Mitochondrion 37%
• Reactive Oxygen Species 25%
• Bcl-2 25%
• Protein Family 12%
• Cell Death 12%
• Caspase 3 12%
• Mitogen-Activated Protein Kinase 12%
• Membrane Potential 12%
• Enhancer Region 12%
• Caspase 12%