Uric acid excretion increases during propofol anesthesia

Akira Masuda*, Takehisa Asahi, Midori Sakamaki, Katsuhito Nakamaru, Koki Hirota, Yusuke Ito

*Corresponding author for this work

Research output: Contribution to journalArticlepeer-review

28 Scopus citations

Abstract

We compared the effect of propofol with that of sevoflurane anesthesia on uric acid (UA) excretion in ASA physical status I and II patients with normal renal function. A propofol group (n = 11) received propofolnitrous oxide-fentanyl after induction of anesthesia by propofol, while a sevoflurane group (n = 12) received sevoflurane-nitrous oxide-fentanyl after induction of anesthesia by thiamylal. UA, creatinine (Cr), and urea nitrogen concentrations in serum and urine were measured before induction of anesthesia, 1, 2, and 3 h after induction, and on Postoperative Day 1. N- acetyl-β-D-glucosaminidase, β2-microglobulin concentrations, and pH in urine were also examined. Plasma clearance of UA (CUA) and Cr (CCr) were calculated. The hourlyconcentration and excretion of urine UA were significantly higher than those of the sevoflurane group (P < 0.01). Significant correlations were noted between the hourly urine volume and UA concentration (r = 0.58, P4 < 0.01 for the propofol group; r = 0.51, P < 0.01 for the sevoflurane group). The CUA of the propofol group was significantly higher than that of the sevoflurane group (22.9 ± 10.6 vs 5.9 ± 3.4 mL/min, mean ± SD, P <0.05). There were no significant differences in other renal variables between the two groups. The present study demonstrated that the UA excretion increased during propofol anesthesia, while it remained stable during sevoflurane anesthesia.

Original languageEnglish
Pages (from-to)144-148
Number of pages5
JournalAnesthesia and Analgesia
Volume85
Issue number1
DOIs
StatePublished - 1997

ASJC Scopus subject areas

  • Anesthesiology and Pain Medicine

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