Kawasaki disease patients homozygous for the rs12252-c variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions

Neil E. Bowles; Cammon B. Arrington; Keiichi Hirono; Tsuneyuki Nakamura; Long Ngo; Yin Shen Wee; Fukiko Ichida; John H. Weis

doi:10.1002/mgg3.79

Kawasaki disease patients homozygous for the rs12252-c variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions

Neil E. Bowles^*, Cammon B. Arrington, Keiichi Hirono, Tsuneyuki Nakamura, Long Ngo, Yin Shen Wee, Fukiko Ichida, John H. Weis

^*Corresponding author for this work

Itoigawa Community Medical Support Unit

Research output: Contribution to journal › Letter › peer-review

3 Scopus citations

Original language	English
Pages (from-to)	356-361
Number of pages	6
Journal	Molecular Genetics and Genomic Medicine
Volume	2
Issue number	4
DOIs	https://doi.org/10.1002/mgg3.79
State	Published - 2014/07

ASJC Scopus subject areas

Molecular Biology
Genetics
Genetics(clinical)

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10.1002/mgg3.79

Cite this

Bowles, N. E., Arrington, C. B., Hirono, K., Nakamura, T., Ngo, L., Wee, Y. S., Ichida, F., & Weis, J. H. (2014). Kawasaki disease patients homozygous for the rs12252-c variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions. Molecular Genetics and Genomic Medicine, 2(4), 356-361. https://doi.org/10.1002/mgg3.79

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title = "Kawasaki disease patients homozygous for the rs12252-c variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions",

author = "Bowles, {Neil E.} and Arrington, {Cammon B.} and Keiichi Hirono and Tsuneyuki Nakamura and Long Ngo and Wee, {Yin Shen} and Fukiko Ichida and Weis, {John H.}",

year = "2014",

month = jul,

doi = "10.1002/mgg3.79",

language = "英語",

volume = "2",

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Bowles, NE, Arrington, CB, Hirono, K, Nakamura, T, Ngo, L, Wee, YS, Ichida, F & Weis, JH 2014, 'Kawasaki disease patients homozygous for the rs12252-c variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions', Molecular Genetics and Genomic Medicine, vol. 2, no. 4, pp. 356-361. https://doi.org/10.1002/mgg3.79

Kawasaki disease patients homozygous for the rs12252-c variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions. / Bowles, Neil E.; Arrington, Cammon B.; Hirono, Keiichi et al.
In: Molecular Genetics and Genomic Medicine, Vol. 2, No. 4, 07.2014, p. 356-361.

Research output: Contribution to journal › Letter › peer-review

TY - JOUR

T1 - Kawasaki disease patients homozygous for the rs12252-c variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions

AU - Bowles, Neil E.

AU - Arrington, Cammon B.

AU - Hirono, Keiichi

AU - Nakamura, Tsuneyuki

AU - Ngo, Long

AU - Wee, Yin Shen

AU - Ichida, Fukiko

AU - Weis, John H.

PY - 2014/7

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AN - SCOPUS:84983206196

SN - 2324-9269

VL - 2

SP - 356

EP - 361

JO - Molecular Genetics and Genomic Medicine

JF - Molecular Genetics and Genomic Medicine

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