Projects per year
Personal profile
Campus career
和漢医薬学総合研究所 准教授 2012/04/01-2017/03/31
和漢医薬学総合研究所 教授 2017/04/01-2019/09/30
薬学・和漢系 教授 2019/10/01-
和漢医薬学総合研究所 教授 2019/10/01-
和漢医薬学総合研究所 教授 2017/04/01-2019/09/30
薬学・和漢系 教授 2019/10/01-
和漢医薬学総合研究所 教授 2019/10/01-
Research interests
病態における自然免疫応答の役割解明と創薬への応用
1)NK細胞のバイオロジーと腫瘍免疫応答における役割
2)がん病態進展における腫瘍微小環境での自然免疫応答の役割
3)がんモデルにおける病態のリアルタイムイメージング
4) 和漢薬による免疫疾患制御
1)NK細胞のバイオロジーと腫瘍免疫応答における役割
2)がん病態進展における腫瘍微小環境での自然免疫応答の役割
3)がんモデルにおける病態のリアルタイムイメージング
4) 和漢薬による免疫疾患制御
Expertise related to UN Sustainable Development Goals
In 2015, UN member states agreed to 17 global Sustainable Development Goals (SDGs) to end poverty, protect the planet and ensure prosperity for all. This person’s work contributes towards the following SDG(s):
Research Fields, Keywords
- Cancer
- Immunology
- Inflammation
- metastasis
- Host defenses
- Natural Killer cell
Field of expertise (Grants-in-aid for Scientific Research classification)
- Oncology
Fingerprint
Dive into the research topics where Yoshihiro Hayakawa is active. These topic labels come from the works of this person. Together they form a unique fingerprint.
- 1 Similar Profiles
Collaborations and top research areas from the last five years
Recent external collaboration on country/territory level. Dive into details by clicking on the dots or
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Research on the role of tissue-resident NK cells in cancer immune-surveillance
Hayakawa, Y. (PI)
2021/04/01 → 2025/03/31
Project: Research
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the effects of CD206-positive macrophages on obesity-associated liver tumors
Usui, I. (PI), Nakagawa, T. (CoPI) & Hayakawa, Y. (CoI)
2016/04/01 → 2019/03/31
Project: Research
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がん悪性化に関わる炎症を制御する生理活性物質の探索
Banbury, L. L. (PI) & Hayakawa, Y. (CoI)
2014/04/25 → 2015/03/31
Project: Research
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CAMSAP2 enhances lung cancer cell metastasis by mediating RASAL2 degradation
Singharajkomron, N., Yodsurang, V., Limprasutr, V., Wattanathamsan, O., Iksen, I., Hayakawa, Y. & Pongrakhananon, V., 2024/02/01, In: Life Sciences. 338, 122391.Research output: Contribution to journal › Article › peer-review
1 Scopus citations -
CAMSAP3-mediated regulation of HMGB1 acetylation and subcellular localization in lung cancer cells: Implications for cell death modulation
Singharajkomron, N., Seephan, S., Iksen, I., Chantaravisoot, N., Wongkongkathep, P., Hayakawa, Y. & Pongrakhananon, V., 2024/06, In: BBA - General Subjects. 1868, 6, 130614.Research output: Contribution to journal › Article › peer-review
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Corrigendum to “CAMSAP3-mediated regulation of HMGB1 acetylation and subcellular localization in lung cancer cells: Implications for cell death modulation”. [BBA - General Subjects (2024) 1868: 130614] (BBA - General Subjects (2024) 1868(6), (S0304416524000576), (10.1016/j.bbagen.2024.130614))
Singharajkomron, N., Seephan, S., Iksen, I., Chantaravisoot, N., Wongkongkathep, P., Hayakawa, Y. & Pongrakhananon, V., 2024/08, In: BBA - General Subjects. 1868, 8, 130644.Research output: Contribution to journal › Comment/debate
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Immunological Aspects of Cancer Cell Metabolism
Ucche, S. & Hayakawa, Y., 2024/05, In: International Journal of Molecular Sciences. 25, 10, 5288.Research output: Contribution to journal › Review article › peer-review
Open Access4 Scopus citations -
Targeting Alpha7 Nicotinic Acetylcholine Receptors in Lung Cancer: Insights, Challenges, and Therapeutic Strategies
Arunrungvichian, K., Vajragupta, O., Hayakawa, Y. & Pongrakhananon, V., 2024/01/12, In: ACS Pharmacology and Translational Science. 7, 1, p. 28-41 14 p.Research output: Contribution to journal › Review article › peer-review
Prizes
Activities
- 27 Oral presentation
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Resveratrol induces apoptosis in melanoma cells via downregulation of survivin expression.
Hayakawa, Y. (Speaker)
2013/10/03 → 2013/10/05Activity: Talk or presentation › Oral presentation
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Role of cross- talk between host immune responses and cancer cells in metastasis disease
Hayakawa, Y. (Speaker)
2013/10/03 → 2013/10/05Activity: Talk or presentation › Oral presentation
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Identification of plant extracts sensitizing breast cancer cells to TRAIL
Hayakawa, Y. (Speaker)
2013/10/03 → 2013/10/05Activity: Talk or presentation › Oral presentation
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Mesenchymal-transitioned cancer cells instigate surrounding cancer cells to metastasis via secretion of WNT ligands
Hayakawa, Y. (Speaker)
2013/10/03 → 2013/10/05Activity: Talk or presentation › Oral presentation
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Cross-talk between host and cancer cells amplify inflammation triggered by gdT17 cells to promote malignant progression.
Hayakawa, Y. (Speaker)
2013/10/03 → 2013/10/05Activity: Talk or presentation › Oral presentation